What describes the pathophysiology of lattice degeneration?

Lattice degeneration is characterized by specific changes in the retinal structure, particularly in the peripheral regions. The condition involves focal retinal thinning, which is a central aspect of its pathophysiology. This thinning occurs due to the various changes in the retina that lead to the formation of atrophic regions. Additionally, there is associated vitreous liquefaction overlying these areas, which means that the gel-like substance inside the eye becomes more fluid and less gel-like, contributing to the changes observed in the retina.

The presence of vitreous liquefaction can lead to increased traction on the retina, which may subsequently predispose the individual to retinal tears or detachment. However, lattice degeneration itself does not equate to complete retinal detachment; instead, it is a precursor condition that may create a risk for such complications if left unmanaged. Understanding this aspect is crucial for preventive strategies in patients with lattice degeneration, as close monitoring can help mitigate the risk of progression to more severe retinal issues.